Hydrogenology
Hydrogenology editorial study record

Hydrogen suppresses oxidative stress by inhibiting the p38 MAPK signaling pathway in preeclampsia

Guo L, Liu M, Duan T. · Adv Clin Exp Med. 2023;32(3):357–367.

PreclinicalOther formsPublished 2023
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized controlled rat study

Research topic

Women’s health

Administration classification

Other forms

Study result signal

Positive preclinical signal with mechanistic co-intervention.

Outcome type

Physiological, tissue and biomarker outcomes in a rat model.

Reported in the source

Methods at a glance

Population or model

Pregnant Sprague-Dawley rats in an L-NAME preeclampsia model.

Sample

Five experimental groups; group sizes were checked in the full methods.

Duration

Late-gestation experimental protocol.

Intervention

Hydrogen-rich saline in the L-NAME intervention arms, with a p38 inhibitor in one additional arm.

Hydrogen form

H₂ dissolved in saline.

H₂ specification

L-NAME was delivered in hydrogen-rich saline; the full article describes preparation and arm-specific dosing.

H₂ flow

Not applicable — saline administration.

O₂ delivered with H₂

No oxygen was co-delivered.

Comparator

Non-pregnant, normal-pregnancy and L-NAME saline controls.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Blood pressure, proteinuria, placental oxidative and inflammatory markers and p38 MAPK expression.

Reported result

The hydrogen-rich-saline arm had lower proteinuria and several placental oxidative, inflammatory and p38-pathway measures than the L-NAME model arm.

Results-extraction completeness

The full article methods, intervention, outcomes, positive and null findings, funding and conflict statements were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Animal-model evidence only; the inhibitor arm is a mechanistic co-intervention and does not establish clinical benefit.

Applies directly to

The reported L-NAME rat model only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Randomization was reported, but concealment and assessor blinding were not established and several outcomes were analyzed.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 36330842 · DOI: 10.17219/acem/154623

Publisher access

A free publisher or repository full article was checked.

Extraction basis

Publisher or repository full article and bibliographic record; checked 10 August 2026.

Record revision

2026-08-10