Hydrogenology
Hydrogenology editorial study record

Association between hydrogen gas inhalation and cardiac output in an asphyxiated piglet model

Sakamoto K, Nakamura S, Tsuchiya T, Mitsuie T, Nakao Y, Htun Y, Yokota T, Inoue K, Inoue E, Wakabayashi T, Morimoto A, Koyano K, Konishi Y, Iwase T, Horii T, Kusaka T. · Scientific Reports. 2026;16:4344.

PreclinicalOther formsPublished 2026
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized two-group newborn-piglet hypoxic-ischemic resuscitation experiment

Research topic

Critical care and ischemia-reperfusion

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Piglets no older than 24 hours subjected to approximately 40 minutes of controlled hypoxic-ischemic insult and resuscitation.

Sample

17 piglets randomized after resuscitation: hypoxic-ischemic control n=10 and H₂ inhalation n=7; cardiac-troponin analysis was available for 9 and 4 animals, respectively.

Duration

Six hours after hypoxic-ischemic insult and resuscitation.

Intervention

Mechanically ventilated H₂ for six hours beginning after resuscitation; concentration varied from 2.1% to 2.7% according to each animal's oxygen requirement.

Hydrogen form

H₂ alone supplied from a 3.8% H₂/96.2% N₂ cylinder and mixed separately with 100% O₂ — not Brown's gas or oxyhydrogen.

H₂ specification

Delivered H₂ 2.1–2.7%; FiO₂ varied from 0.21 to 0.40. Concentrations were adjusted through two gas sources and H₂ was monitored, but absolute total flow was not stated.

H₂ flow

Not reported in mL/min — concentration and ventilator settings were provided without an absolute minute-flow value.

O₂ delivered with H₂

O₂ was supplied separately to achieve FiO₂ 0.21–0.40; absolute O₂ flow in mL/min was not reported.

Comparator

The same hypoxic-ischemic insult and mechanical ventilation without H₂ therapy.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Serial left/right stroke volume and cardiac output, RV outflow velocity-time integral, blood pressure/heart rate/blood gases and cardiac troponin T.

Reported result

Right-ventricular cardiac-output area under the curve and the five-hour value favored H₂; six-hour troponin T was lower in the small available subset. Left cardiac output, pulmonary-artery acceleration time, blood gases and several time-point comparisons did not differ significantly.

Results-extraction completeness

The complete free PMC article, figures, methods and disclosures were checked for randomization, variable H₂/O₂ composition, missing absolute flows, positive and null cardiac findings, reduced troponin sample, funding and conflicts.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Very small neonatal-piglet experiment, unequal groups, short follow-up, anesthesia/ventilation confounding, possible unmeasured shunts, many serial comparisons and troponin available for only four H₂ animals. Japanese public/university grants funded the study; authors declared no competing interests.

Applies directly to

Experimental neonatal asphyxia in piglets; it does not establish cardiovascular or neurologic treatment in human newborns.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 41530398 · DOI: 10.1038/s41598-026-35115-2

Publisher access

Free full article in PubMed Central.

Extraction basis

Complete PMC article and PubMed metadata; full-text extraction checked 9 August 2026.

Record revision

2026-08-10