Hydrogenology
Hydrogenology editorial study record

Hydrogen improves neurological function through attenuation of blood-brain barrier disruption in spontaneously hypertensive stroke-prone rats.

Takeuchi S, Nagatani K, Otani N, Nawashiro H, Sugawara T, Wada K, Mori K. · 2015.

PreclinicalH₂-rich waterPublished 2015
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized controlled rat study

Research topic

Other neurological conditions

Administration classification

H₂-rich water

Study result signal

Mixed positive preclinical signal.

Outcome type

Animal survival, functional, tissue and biomarker outcomes.

Reported in the source

Methods at a glance

Population or model

Spontaneously hypertensive stroke-prone rats.

Sample

56 rats: hydrogen-rich-water n=28 and control-water n=28.

Duration

Long-term follow-up through neurological and survival assessment.

Intervention

Long-term ingestion of hydrogen-rich water.

Hydrogen form

H₂ dissolved in drinking water.

H₂ specification

Preparation and replacement schedule were checked in the full article.

H₂ flow

Not applicable.

O₂ delivered with H₂

No oxygen was co-delivered.

Comparator

Regular drinking water.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Neurological function, survival, cerebral bleeding/infarction, blood-brain-barrier leakage, oxidative markers and MMP-9.

Reported result

Neurological function and several tissue or barrier measures favored hydrogen water, while the survival difference was not statistically significant.

Results-extraction completeness

The full article was checked for methods, intervention details, outcomes, positive and null findings, funding and conflicts. This record does not imply medical efficacy.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Animal model with multiple outcomes; the null survival comparison must remain distinct from favorable surrogate measures.

Applies directly to

The reported stroke-prone rat model only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Random assignment was reported, but concealment and blinded outcome assessment were not fully established.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 25925889 · DOI: 10.1186/s12868-015-0165-3

Publisher access

A free full article is available through PubMed Central.

Extraction basis

Official PubMed Central full article and PubMed bibliographic record; source identity, methods, intervention, results, funding and conflicts checked 10 August 2026.

Record revision

2026-08-10