Hydrogenology
Hydrogenology editorial study record

Hydrogen-rich saline reduces cell death through inhibition of DNA oxidative stress and overactivation of PARP-1 in retinal ischemia-reperfusion injury

Liu Y et al. · Mol Med Rep. 2015;12(2):2495–2502.

PreclinicalOther formsPublished 2015
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat retinal ischemia-reperfusion experiment

Research topic

Eye health

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Rats with retinal ischemia-reperfusion injury induced by transient elevation of intraocular pressure

Sample

Group-level histology assays commonly report n=6; the total number of animals was not clearly stated in the extracted article text

Duration

Daily treatment from reperfusion until the scheduled tissue assessments

Intervention

Hydrogen-rich saline, 5 mL/kg intraperitoneally once daily from reperfusion until sacrifice

Hydrogen form

H₂ dissolved in saline — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

The introduction discusses theoretical saturation up to 0.8 mM; the actual preparation concentration was not clearly stated in the extracted methods.

H₂ flow

Not applicable — intraperitoneal saline, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

Ischemia-reperfusion animals receiving non-hydrogen saline, with sham-operated controls

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Retinal morphology, cell loss, TUNEL staining, 8-OHdG, PARP-1 and caspase-3

Reported result

The article reports less retinal cell loss, DNA oxidative-stress signal, PARP-1 overactivation and caspase-3 activation with hydrogen-rich saline.

Results-extraction completeness

Methods and principal laboratory outcomes checked in the free full text; exact total animal allocation and actual saline H₂ concentration remain unclear.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Animal injury model with laboratory and histologic outcomes; total animal allocation and actual H₂ concentration require further clarification. It cannot establish benefit in people.

Applies directly to

Experimental retinal ischemia-reperfusion injury in rats, not human eye disease.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 25954991 · DOI: 10.3892/mmr.2015.3731

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC full text and PubMed bibliographic record.

Record revision

2026-08-10