Hydrogenology
Hydrogenology editorial study record

Suppression of ERK phosphorylation through oxidative stress is involved in the mechanism underlying sevoflurane-induced toxicity in the developing brain.

Yufune S, Satoh Y, Akai R, Yoshinaga Y, Kobayashi Y, Endo S, Kazama T. · 2016.

PreclinicalOther formsPublished 2016
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled neonatal-mouse mechanistic study

Research topic

Other neurological conditions

Administration classification

Other forms

Study result signal

Positive preclinical mechanistic signal.

Outcome type

Animal molecular, cell-death and behavioral outcomes.

Reported in the source

Methods at a glance

Population or model

Developing mice exposed to sevoflurane and related pathway interventions.

Sample

Animal groups and assay-specific counts were checked in the full article.

Duration

Acute neonatal exposure with later behavioral assessment.

Intervention

Molecular hydrogen administered during the sevoflurane neurotoxicity protocol.

Hydrogen form

Article-specified molecular H₂ exposure.

H₂ specification

Exposure details were checked in the full methods; no clinical dose is inferred.

H₂ flow

Not reported as a patient-delivery flow.

O₂ delivered with H₂

Carrier conditions are reported in the experimental methods.

Comparator

Sevoflurane, pathway-inhibitor and hydrogen intervention comparisons.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

ERK phosphorylation, neuronal apoptosis and later behavioral measures.

Reported result

Hydrogen restored ERK phosphorylation and attenuated sevoflurane-associated apoptosis in the reported developing-brain model.

Results-extraction completeness

The full article was checked for methods, intervention details, outcomes, positive and null findings, funding and conflicts. This record does not imply medical efficacy.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Mechanistic animal study with multiple pathway interventions; it does not establish pediatric anesthetic benefit.

Applies directly to

The reported neonatal-mouse sevoflurane model only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Controlled interventions were used, but concealment, assessor blinding and multiplicity handling were unclear.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 26905012 · DOI: 10.1038/srep21859

Publisher access

A free full article is available through PubMed Central.

Extraction basis

Official PubMed Central full article and PubMed bibliographic record; source identity, methods, intervention, results, funding and conflicts checked 10 August 2026.

Record revision

2026-08-10