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Hydrogenology editorial study record

Hydrogen-rich saline promotes microglia M2 polarization and complement-mediated synapse loss to restore behavioral deficits following hypoxia-ischemic injury in neonatal mice via AMPK activation

Chu X et al. · J Neuroinflammation. 2019;16:104.

PreclinicalOther formsPublished 2019
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled neonatal-mouse hypoxia-ischemia experiment plus BV-2 microglial-cell experiments

Research topic

Other neurological conditions

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Postnatal-day-7 mouse pups with Rice-Vannucci hypoxia-ischemia injury and cultured BV-2 cells

Sample

Multiple cohorts: principal tissue assays n=4–6/group and behavioral cohorts n=10/group; the article does not present a single total animal count

Duration

Treatment on post-injury days 1–3, with acute tissue and later behavioral and synaptic assessments

Intervention

Hydrogen-rich saline 5 mL/kg intraperitoneally on days 1, 2 and 3 after hypoxia-ischemia, with additional AMPK-inhibitor experiments

Hydrogen form

H₂ dissolved in saline or culture medium — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

>0.6 mM dissolved H₂; saline was prepared for 12 hours at 0.4 MPa and concentration was checked with a dissolved-H₂ meter.

H₂ flow

Not applicable — saline and cell-medium intervention, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the H₂ intervention.

Comparator

Hypoxia-ischemia without H₂, sham controls and AMPK-inhibitor comparison groups

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Inflammatory-cell accumulation, microglial polarization, AMPK and NF-κB, neurological behavior, synaptic proteins and complement components

Reported result

The article reports less neuroinflammation, improved behavioral and synaptic measures and changes consistent with AMPK activation; an AMPK inhibitor reversed several reported effects.

Results-extraction completeness

The complete open-access article and supplementary cohort descriptions were checked for dose, concentration, allocation by assay, schedules, positive and null outcomes, funding and conflicts.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Complex neonatal mouse and cell study with multiple mechanistic groups and outcomes. It does not establish treatment benefit after human neonatal hypoxic-ischemic injury.

Applies directly to

Neonatal hypoxia-ischemia in mice and cultured microglia.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 31103039 · DOI: 10.1186/s12974-019-1488-2

Publisher access

Open-access full text is available through PMC and the publisher.

Extraction basis

Open-access publisher/PMC full text and PubMed record.

Record revision

2026-08-10