Hydrogenology
Hydrogenology editorial study record

Hydrogen-rich saline ameliorates hippocampal neuron apoptosis through up-regulating cystathionine β-synthase after cerebral ischemia-reperfusion in rats

Cong HM et al. · Iran J Basic Med Sci. 2020;23(4):494–499.

PreclinicalOther formsPublished 2020
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized three-group rat middle-cerebral-artery-occlusion experiment

Research topic

Other neurological conditions

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Adult male Sprague-Dawley rats after two hours of MCAO and reperfusion

Sample

36 rats, n=12/group

Duration

Two hours of occlusion followed by reperfusion; principal assessment at 24 hours

Intervention

Hydrogen-rich saline 1 mL/kg after the beginning of reperfusion

Hydrogen form

H₂ dissolved in saline — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

At least 0.6 mmol/L dissolved H₂; 1 mL/kg. Administration route was not stated in the article’s experiment-design paragraph.

H₂ flow

Not applicable — saline administration, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the H₂ intervention.

Comparator

Untreated ischemia-reperfusion and sham surgery

Reported, not endorsed

Outcomes and reported result

Outcomes measured

H₂S, S100β, NSE, ROS, MDA, SOD, hippocampal histology and apoptosis, CBS, Nrf2 and HO-1

Reported result

The article reports higher H₂S, CBS, Nrf2, HO-1 and SOD and lower injury, apoptosis, S100β, NSE, ROS and MDA with H₂-rich saline.

Results-extraction completeness

Allocation, concentration threshold, dose, timing, methods and principal outcomes checked in the open-access full text; route is not reported.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Small acute preclinical stroke model with many molecular outcomes and an unreported administration route.

Applies directly to

Cerebral ischemia-reperfusion in rats, not stroke care in people.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 32489564 · DOI: 10.22038/ijbms.2020.41751.9857

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC article and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10