Hydrogenology
Hydrogenology editorial study record

Inhalation of hydrogen gas protects against mitomycin-induced pulmonary veno-occlusive disease

Zhang C et al. · Respir Res. 2024;25:281.

PreclinicalOther formsPublished 2024
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized four-group rat prevention-and-treatment experiment

Research topic

Respiratory health

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Female Sprague-Dawley rats with mitomycin-C-induced pulmonary veno-occlusive disease

Sample

60 rats, n=15/group; smaller prespecified subsets were used for hemodynamic, imaging and tissue assays

Duration

One hour twice daily for either 28 days (prevention) or 14 days (delayed treatment)

Intervention

42% H₂ mixture for 1 hour twice daily, started either with mitomycin C for 28 days or after 14 days for a further 14 days

Hydrogen form

Diluted oxyhydrogen: Brown’s gas was generated first, then diluted with nitrogen to 42% H₂, 21% O₂ and 37% N₂.

H₂ specification

Final chamber mixture: 42% H₂, 21% O₂ and 37% N₂; total flow 3.8 L/min.

H₂ flow

1,596 mL/min H₂ — calculated from the reported 42% H₂ in a 3.8 L/min total mixture.

O₂ delivered with H₂

798 mL/min O₂ — calculated from the reported 21% O₂ in the 3.8 L/min total mixture.

Comparator

Normal-air control and mitomycin-C disease control without H₂

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Survival, pulmonary hemodynamics, right-heart function and hypertrophy, vascular remodeling, inflammation, oxidative stress and EndoMT markers

Reported result

The article reports improved hemodynamic, right-heart, vascular, inflammatory and oxidative measures in both H₂ schedules.

Results-extraction completeness

Allocation, gas generation, final mixture, total flow, methods, figures, funding and conflicts checked in the open-access full text. One figure caption prints 33% N₂, while the methods state 37% and sum to 100%.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Chemotherapy-induced rat model, repeated high-concentration gas exposure and multiple mechanistic outcomes. Calculated component flows are not direct flow-meter readings for each gas.

Applies directly to

Mitomycin-C-induced pulmonary veno-occlusive disease in rats, not human pulmonary hypertension.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 39014440 · DOI: 10.1186/s12931-024-02906-y

Publisher access

Open-access full text is available through PMC and the publisher.

Extraction basis

Open-access publisher/PMC full text and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10