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Hydrogen gas reduced acute-hyperglycemia-enhanced hemorrhagic transformation in a focal-ischemia rat model

Chen CH et al. · Neuroscience. 2010;169(1):402–414.

PreclinicalInhaled H₂Published 2010
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat hyperglycemia plus transient middle-cerebral-artery-occlusion experiment

Research topic

Other neurological conditions

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

72 Sprague-Dawley rats receiving dextrose and 90-minute focal cerebral ischemia

Sample

72 rats across sham, sham plus H₂, ischemia and ischemia plus H₂ groups

Duration

One or two hours of inhalation during reperfusion; outcomes at 24 hours

Intervention

Premixed gas inhalation for 1 or 2 hours immediately after ischemia

Hydrogen form

H₂ diluted with O₂ and N₂ — not Brown’s gas.

H₂ specification

2.9% H₂ + 21% O₂ + 76.1% N₂.

H₂ flow

Not reported — premixed gas composition is given, but H₂ flow in mL/min is not.

O₂ delivered with H₂

21% O₂ was co-delivered; O₂ flow in mL/min was not reported.

Comparator

Corresponding premixed gas without H₂ and sham groups

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Infarct and hemorrhage volumes, neurological score, glucose, oxidative markers, MMP-2/MMP-9 and blood-brain-barrier proteins

Reported result

Two-hour H₂ inhalation reduced hemorrhage and infarct volumes and improved neurological scores; one hour reduced hemorrhage but not infarct volume. MMP-9 and several oxidative markers decreased, while MMP-2, ZO-1, occludin, collagen IV and AQP4 did not significantly change.

Results-extraction completeness

Gas composition, allocation, methods, positive and null outcomes, funding and figures checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Acute rat stroke model with experimentally induced hyperglycemia and multiple laboratory outcomes; no human participants.

Applies directly to

Transient focal cerebral ischemia with acute hyperglycemia in rats.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 20423721 · DOI: 10.1016/j.neuroscience.2010.04.043

Publisher access

Free full text is available through PMC.

Extraction basis

Open-access PMC article and PubMed record.

Record revision

2026-08-10