Hydrogenology
Hydrogenology editorial study record

Molecular hydrogen reduces LPS-induced neuroinflammation and promotes recovery from sickness behaviour in mice

Spulber S et al. · PLoS One. 2012;7(7):e42078.

PreclinicalH₂-rich waterPublished 2012
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled mouse endotoxin experiment with behavioral, inflammatory and gene-expression cohorts

Research topic

Immune and inflammatory conditions

Administration classification

H₂-rich water

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Mice receiving lipopolysaccharide to induce systemic inflammation and sickness behavior

Sample

Behavioral cohorts included sham/control n=15, sham/H₂ n=15, LPS/control n=13 and LPS/H₂ n=14; additional home-cage and tissue cohorts were smaller

Duration

Seven-day preadministration followed by continued intake during behavioral and tissue follow-up

Intervention

Hydrogen-enriched electrochemically reduced drinking water ad libitum, started 7 days before LPS and continued afterward

Hydrogen form

H₂ dissolved in drinking water — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

Drinking water contained 510.8 ± 21.9 ppb dissolved H₂; H₂-rich cell medium contained 78.5 ± 20.1 ppb.

H₂ flow

Not applicable — oral drinking water, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

Ordinary drinking water in sham-treated and LPS-treated mice

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Body weight, spontaneous and novel-environment behavior, brain and peripheral cytokines and inflammatory gene expression

Reported result

The article reports less sickness behavior and faster behavioral recovery. Baseline TNF-α decreased and IL-10 increased; the neuroinflammatory response was described as larger in amplitude but shorter in duration.

Results-extraction completeness

The complete open-access article was checked for water characterization, allocation, behavioral and molecular outcomes, funding and conflicts.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Mouse endotoxin model with multiple behavioral and molecular cohorts. An employee of Nihon-Trim was an author and the company funded the study.

Applies directly to

LPS-induced sickness and neuroinflammation in mice, not a clinical neurological or infectious disease.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 22860058 · DOI: 10.1371/journal.pone.0042078

Publisher access

Open-access full text is available through PMC and PLOS.

Extraction basis

Open-access PMC/PLOS full text and PubMed record.

Record revision

2026-08-10