Hydrogenology
Hydrogenology editorial study record

Suppressive Effect of High Hydrogen Generating High Amylose Cornstarch on Subacute Hepatic Ischemia-reperfusion Injury in Rats.

Tanabe H, Sasaki Y, Yamamoto T, Kiriyama S, Nishimura N. · Bioscience of Microbiota, Food and Health. 2012;31:103-108.

PreclinicalOther formsPublished 2012
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat study

Research topic

Liver health

Administration classification

Other forms

Study result signal

Mixed preclinical signal.

Outcome type

Animal liver tissue and biomarker outcomes.

Reported in the source

Methods at a glance

Population or model

Male Sprague-Dawley rats in a hepatic ischemia-reperfusion model.

Sample

49 rats across four diet and surgery groups of 12 or 13.

Duration

14-day diet followed by 48-hour reperfusion.

Intervention

High-amylose cornstarch diet used to increase endogenous intestinal hydrogen production for 14 days before surgery.

Hydrogen form

Endogenous intestinal H2 generated through fermentation; not Brown's gas.

H₂ specification

Breath hydrogen was reported at about ten times and portal-vein hydrogen at about five times control values.

H₂ flow

Not applicable — dietary fermentation.

O₂ delivered with H₂

No oxygen was co-delivered.

Comparator

Control diet with sham or ischemia-reperfusion surgery.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

AST, ALT, liver histology, myeloperoxidase and oxidative-stress measures.

Reported result

AST was lower with the high-amylose diet; ALT showed a non-significant trend, and several oxidative and inflammatory markers were null.

Results-extraction completeness

The full article, methods, tables and positive and null findings were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Dietary intervention has effects beyond hydrogen production, and most measured markers did not show a clear benefit.

Applies directly to

The reported rat hepatic ischemia-reperfusion model.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Unclear allocation concealment and assessor blinding; dietary co-effects are important.

Appraisal domains

Randomization/allocation reporting is limited · Blinding of personnel is unclear · Attrition is incompletely reported in places · Blinding of outcome assessors is unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 24936356 · DOI: 10.12938/bmfh.31.103

Publisher access

Free full article in PubMed Central.

Extraction basis

PubMed record and PubMed Central full text; extraction checked 9 August 2026.

Record revision

2026-08-10