Hydrogenology
Hydrogenology editorial study record

Hydrogen-rich saline alleviates inflammation and apoptosis in myocardial ischemia-reperfusion injury via PINK-mediated autophagy

Yao L et al. · Int J Mol Med. 2019;44(3):1048–1062.

PreclinicalOther formsPublished 2019
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat myocardial ischemia-reperfusion experiment plus H9C2 hypoxia-reoxygenation cell experiments

Research topic

Cardiovascular and circulatory health

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Rats with 30 minutes of myocardial ischemia and 24 hours of reperfusion; cultured H9C2 cardiomyoblasts

Sample

24 rats randomized to four groups, n=6 per group; cell experiments used separate repeated comparisons

Duration

Single pre-reperfusion injection; principal in-vivo assessment after 24 hours of reperfusion

Intervention

0.6 mmol/L hydrogen-rich saline, 10 mL/kg intraperitoneally 5 minutes before reperfusion; cells received 0.6 mmol/L H₂-rich medium

Hydrogen form

H₂ dissolved in saline or culture medium — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

0.6 mmol/L dissolved H₂.

H₂ flow

Not applicable — saline injection and culture medium, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the H₂ intervention.

Comparator

Control and ischemia-reperfusion groups receiving ordinary saline; corresponding cell-culture controls

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Infarct size, cardiac function, cTnI, CK-MB, cytokines, apoptosis, cell viability and PINK1/Parkin autophagy markers

Reported result

The article reports smaller infarcts and favorable cardiac-function, inflammatory, apoptotic and cell-viability measures with H₂. PINK1 pathway manipulation attenuated several reported effects.

Results-extraction completeness

Dose, allocation, methods, figures and principal results checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical study combining a small rat cohort and cultured cells, with multiple mechanistic outcomes and no human participants.

Applies directly to

Experimental myocardial ischemia-reperfusion in rats and hypoxia-reoxygenation in cultured cells.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 31524220 · DOI: 10.3892/ijmm.2019.4264

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC full text and PubMed record.

Record revision

2026-08-10