Hydrogenology
Hydrogenology editorial study record

Hydrogen in Patients With Corticosteroid-Refractory/Dependent Chronic Graft-Versus-Host-Disease: A Single-Arm, Multicenter, Open-Label, Phase 2 Trial

Qian L, Liu M, Shen J, Cen J, Zhao D. · Frontiers in Immunology. 2020;11:598359.

HumanH₂-rich waterPublished 2020
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Human

Reported design

Prospective multicenter, open-label, single-arm phase 2 trial

Research topic

Immune and inflammatory conditions

Administration classification

H₂-rich water

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

People younger than 65 years with corticosteroid-refractory or corticosteroid-dependent chronic graft-versus-host disease after allogeneic stem-cell transplantation.

Sample

24 participants from four institutions; median age 27 years. Nineteen had severe cGVHD and 19 were receiving corticosteroids at entry.

Duration

Planned treatment for up to 12 months, with assessments every 3 months; survival follow-up extended to a median of 38.5 months.

Intervention

Hydrogen-rich water 4 mL/kg orally three times daily as the only new intervention. Background corticosteroids and other immunosuppressants continued and could be tapered under the protocol.

Hydrogen form

H₂ dissolved in drinking water — H₂ only, not Brown's gas.

H₂ specification

The water contained 0.8 ppm dissolved H₂.

H₂ flow

Not applicable — drinking water, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

No concurrent control group; the article discusses historical response rates only.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

NIH organ response criteria, overall response, Karnofsky performance status, prednisone use, toxicity and survival.

Reported result

At last evaluation, 18/24 participants had an objective response (8 complete and 10 partial); five progressed and one was stable. Eighteen completed 12 months, two were lost to follow-up and four died within 12 months. Among responders, median prednisone dose fell from 0.185 to 0 mg/kg/day (p=0.004). The estimated 4-year survival was 74.7%; the responder-versus-nonresponder survival comparison was post hoc and cannot establish treatment causality.

Results-extraction completeness

The complete publisher article, tables, protocol details, organ-level responses, deaths, toxicity, funding, product supply and conflict declaration were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Small uncontrolled open-label study with continuing background immunosuppression, organ-specific denominators and responder-defined survival comparisons. It cannot distinguish H₂ effects from natural history, co-treatment, regression to the mean or selection. Beijing Huoliqingyuan Beverage Co. supplied the water; the study was funded by the National Natural Science Foundation of China (81800180), and authors declared no commercial or financial conflicts.

Applies directly to

Corticosteroid-refractory/dependent cGVHD under this protocol; confirmatory controlled trials are required.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 33324415 · DOI: 10.3389/fimmu.2020.598359

Publisher access

Open-access publisher full article and PubMed Central copy.

Extraction basis

Frontiers publisher full article and PubMed Central copy; full-text extraction checked 8 August 2026.

Record revision

2026-08-10