Hydrogenology
Hydrogenology editorial study record

Hydrogen Attenuates Endotoxin-Induced Lung Injury by Activating Thioredoxin 1 and Decreasing Tissue Factor Expression

Li Q, Hu L, Li J, Yu P, Hu F, Wan B, Xu M, Cheng H, Yu W, Jiang L, Shi Y, Li J, Duan M, Long Y, Liu WT. · 2021.

PreclinicalInhaled H₂Published 2021
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled mouse and cell study

Research topic

Respiratory health

Administration classification

Inhaled H₂

Study result signal

Positive preclinical signal.

Outcome type

Animal survival, tissue and mechanistic outcomes.

Reported in the source

Methods at a glance

Population or model

Mice with endotoxin-induced acute lung injury and related cell experiments.

Sample

Multiple animal cohorts and cell assays; totals vary by experiment.

Duration

Acute and survival follow-up under article-specific schedules.

Intervention

4% hydrogen gas inhalation after endotoxin exposure, with related cell experiments.

Hydrogen form

Inhaled molecular H₂.

H₂ specification

4% H₂; the methods report gas delivery at 400 mL/min for the chamber system.

H₂ flow

400 mL/min total chamber delivery reported; the hydrogen fraction was 4%.

O₂ delivered with H₂

Carrier-gas composition is reported in the full methods; no Brown's-gas classification is inferred.

Comparator

Endotoxin injury without H₂ and laboratory controls.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Survival, lung injury, inflammation, coagulation-related markers and thioredoxin signaling.

Reported result

The article reports higher survival and favorable lung-injury, inflammatory and signaling measures with hydrogen in the tested model.

Results-extraction completeness

The full article was checked for methods, intervention details, outcomes, positive and null findings, funding and conflicts. This record does not imply medical efficacy.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical evidence only; multiple cohorts and mechanistic endpoints.

Applies directly to

The reported endotoxin mouse-lung-injury model only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Controlled groups were used, while concealment, blinding and multiplicity handling were not fully established.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 33767697 · DOI: 10.3389/fimmu.2021.625957

Publisher access

A free full article is available through PubMed Central.

Extraction basis

Official PubMed Central full article and PubMed bibliographic record; source identity, methods, intervention, results, funding and conflicts checked 10 August 2026.

Record revision

2026-08-10