Hydrogenology
Hydrogenology editorial study record

Hydrogen exerts neuroprotective effects by inhibiting oxidative stress in experimental diabetic peripheral neuropathy rats

Han XC, Ye ZH, Hu HJ, Sun Q, Fan DF. · Medical Gas Research. 2023;13(2):72–77.

PreclinicalOther formsPublished 2023
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized three-group streptozotocin-induced diabetes experiment

Research topic

Metabolic and lipid health

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Adult male Sprague-Dawley rats with diabetes induced by a single intraperitoneal 65 mg/kg streptozotocin injection.

Sample

24 rats randomized to healthy control, untreated diabetes and diabetes plus hydrogen-rich saline, n=8 per group.

Duration

Daily treatment for eight weeks.

Intervention

Beginning three days after diabetes induction, rats received hydrogen-rich saline 10 mL/kg by intraperitoneal injection once daily; no insulin or antidiabetic drugs were given.

Hydrogen form

H₂ dissolved in saline — H₂ only, not Brown's gas and not inhalation.

H₂ specification

Hydrogen was dissolved in saline for 3 hours at 0.4 MPa and the solution was prepared one day before use; gas chromatography confirmed a target concentration of 0.6 mmol/L.

H₂ flow

Not applicable — intraperitoneal hydrogen-rich saline, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

Diabetic rats receiving the same 10 mL/kg volume of ordinary physiological saline and non-diabetic controls receiving vehicle.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Body-mass gain, blood glucose, motor nerve-conduction velocity, sciatic-nerve myelin morphology and density, malondialdehyde, reactive oxygen species, 8-hydroxy-2-deoxyguanosine, superoxide dismutase and Nrf2, heme oxygenase-1 and catalase protein expression.

Reported result

Hydrogen-rich saline partially restored motor nerve-conduction velocity and myelinated-nerve density and area and reduced the reported oxidative-stress measures while increasing SOD and Nrf2-pathway proteins versus untreated diabetic rats. Body-mass gain remained non-significantly different from untreated diabetes at weeks 4 and 8 (p>0.05).

Results-extraction completeness

The complete open-access article, allocation, saline preparation, concentration and dose, positive and null results, funding and conflict statement were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical model with only eight rats per group, one sex, many biochemical outcomes and no behavioral pain testing or long-term post-treatment follow-up. The abstract incorrectly contrasts 'hydrogen inhalation' with hydrogen-rich saline, while the Methods describe only daily intraperitoneal hydrogen-rich saline; the structured record follows the Methods and flags the discrepancy. The Innovation Cultivation Foundation of Navy General Hospital funded the work; authors declared no conflicts of interest.

Applies directly to

Streptozotocin-induced diabetic peripheral-nerve injury in rats; it does not establish treatment efficacy, dosing or safety in people with diabetic neuropathy.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 36204786 · DOI: 10.4103/2045-9912.345171

Publisher access

Open-access Medical Gas Research article and PubMed Central copy.

Extraction basis

Complete PubMed Central/Medical Gas Research article and PubMed metadata; full-text extraction checked 9 August 2026.

Record revision

2026-08-10