Hydrogenology
Hydrogenology editorial study record

Inhalation of 4% and 67% hydrogen ameliorates oxidative stress, inflammation, apoptosis, and necroptosis in a rat model of glycerol-induced acute kidney injury.

Xue JL, Liu BY, Zhao M, Zhang MY, Wang MY, Gu QQ, Zhang XY, Qin SC. · 2023.

PreclinicalInhaled H₂Published 2023
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat dose-comparison study

Research topic

Kidney and urinary health

Administration classification

Inhaled H₂

Study result signal

Positive preclinical signal without a consistent dose response.

Outcome type

Animal renal, tissue and mechanistic biomarker outcomes.

Reported in the source

Methods at a glance

Population or model

Rats with glycerol-induced rhabdomyolysis-associated acute kidney injury.

Sample

Animal groups and assay counts were checked in the full article.

Duration

72-hour injury and treatment assessment.

Intervention

Inhalation of 4% or 67% hydrogen after glycerol administration.

Hydrogen form

Inhaled molecular H₂ at low and high concentrations.

H₂ specification

4% and 67% H₂; the high-concentration mixture requires article-specific handling.

H₂ flow

Absolute H₂ flow was not separately reported here.

O₂ delivered with H₂

Carrier-gas composition differs by arm; no unreported oxygen flow is inferred.

Comparator

Normal and glycerol-injury controls plus two H₂ concentrations.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Renal function, histology, oxidative stress, inflammation, apoptosis and necroptosis.

Reported result

Both hydrogen concentrations were reported to improve several renal-injury measures; a consistent dose-response was not observed, although some histology favored 67%.

Results-extraction completeness

The full article was checked for methods, intervention details, outcomes, positive and null findings, funding and conflicts. This record does not imply medical efficacy.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Animal model, many endpoints and no consistent dose-response; very high H₂ concentration limits translation.

Applies directly to

The reported glycerol-induced rat AKI model only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Dose controls were present, but concealment, blinded histology and multiplicity handling were unclear.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 36204787 · DOI: 10.4103/2045-9912.345169

Publisher access

A free full article is available through PubMed Central.

Extraction basis

Official PubMed Central full article and PubMed bibliographic record; source identity, methods, intervention, results, funding and conflicts checked 10 August 2026.

Record revision

2026-08-10