Hydrogenology
Hydrogenology editorial study record

Hydrogen gas alleviates lipopolysaccharide-induced acute lung injury and inflammatory response in mice

Yin H et al. · J Inflamm (Lond). 2022;19:16.

PreclinicalInhaled H₂Published 2022
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized mouse acute-lung-injury experiment plus RAW 264.7 macrophage experiments

Research topic

Respiratory health

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

105 female C57BL/6 mice challenged with lipopolysaccharide and cultured mouse macrophages

Sample

105 mice randomized to three groups of 35; n=10/group for 72-hour survival and the remainder for timed tissue sampling

Duration

Continuous inhalation for 72 hours

Intervention

Continuous 42% H₂ inhalation for 72 hours after LPS or saline administration

Hydrogen form

Diluted oxyhydrogen: a 66.7% H₂/33.3% O₂ Brown’s-gas output was diluted with nitrogen to 42% H₂, 21% O₂ and 37% N₂.

H₂ specification

Final animal mixture: 42% H₂, 21% O₂ and 37% N₂. The generator output was reported as 3 L/min before nitrogen dilution.

H₂ flow

The generator’s reported 3 L/min oxyhydrogen output corresponds to about 2,010 mL/min H₂; final total chamber flow after nitrogen dilution was not reported.

O₂ delivered with H₂

The generator output corresponds to about 990 mL/min O₂ before dilution; the final animal mixture contained 21% O₂, but final O₂ flow was not reported.

Comparator

LPS acute-lung-injury mice without H₂ and H₂-only controls

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Survival, lung histology, cytokines, oxidative measures, TLR4/NF-κB signaling and macrophage-cell outcomes

Reported result

The article reports survival of 80% with LPS plus H₂ versus 60% with LPS, less tissue injury and lower several inflammatory and oxidative measures. Serum IL-6 did not change with treatment.

Results-extraction completeness

Allocation, gas generation and composition, duration, principal positive and null results, funding and conflicts checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Mouse LPS model with continuous very-high-concentration exposure; the study was funded by the device manufacturer. Component flows are calculated from generator output, not measured inspired dose.

Applies directly to

LPS-induced acute lung injury in mice, not acute respiratory distress syndrome in patients.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 36253774 · DOI: 10.1186/s12950-022-00314-x

Publisher access

Open-access full text is available through PMC and the publisher.

Extraction basis

Open-access PMC/publisher full text and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10