Hydrogenology
Hydrogenology editorial study record

Hydrogen-rich saline alleviates cardiomyocyte apoptosis by reducing expression of calpain1 via miR-124-3p

Xue X et al. · ESC Heart Fail. 2023;10(5):3077–3090.

PreclinicalOther formsPublished 2023
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Mouse myocardial ischemia-reperfusion experiment plus neonatal-rat cardiomyocyte experiments

Research topic

Cardiovascular and circulatory health

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Male C57BL/6 mice and cultured neonatal rat cardiomyocytes

Sample

50 mice divided into five groups, 10/group; five/group were used for infarct staining and five/group for stored-tissue analyses

Duration

30 minutes of ischemia followed by 24 hours of reperfusion; multiple cellular schedules

Intervention

0.6 mmol/L hydrogen-rich saline, 10 mL/kg intraperitoneally 5 minutes before reperfusion; cells received 0.6 mmol/L H₂-rich medium

Hydrogen form

H₂ dissolved in saline or culture medium — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

0.6 mmol/L dissolved H₂; saline was saturated for 6 hours at 0.4 MPa and verified by gas chromatography.

H₂ flow

Not applicable — saline and culture medium, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

Ischemia-reperfusion and hypoxia-reoxygenation controls plus pathway-manipulation groups

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Infarct size, cardiac function, apoptosis, cell viability and miR-124-3p/calpain1 pathway

Reported result

The article reports improved cardiac and cell-injury outcomes with H₂-rich saline and attenuation of the effect when the proposed pathway was manipulated.

Results-extraction completeness

The complete open-access article was checked for preparation, dose, animal allocation, intervention timing, figures, funding and conflicts.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical mouse and cell work, many pathway experiments and no human participants. The article declares no conflicts.

Applies directly to

Experimental myocardial ischemia-reperfusion and cultured cardiomyocytes, not patients with myocardial infarction.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 37602925 · DOI: 10.1002/ehf2.14492

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC full text and PubMed record.

Record revision

2026-08-10