Hydrogenology
Hydrogenology editorial study record

Hydrogen ameliorates oxidative stress via PI3K-Akt signaling pathway in UVB-induced HaCaT cells

Zhang B, Zhao Z, Meng X, Chen H, Fu G, Xie K. · Int J Mol Med. 2018;41(6):3653–3661.

PreclinicalOther formsPublished 2018
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled cell-culture study

Research topic

Skin, wound and aging research

Administration classification

Other forms

Study result signal

Positive cell-culture signal.

Outcome type

Cell viability and mechanistic pathway outcomes.

Reported in the source

Methods at a glance

Population or model

HaCaT keratinocytes exposed to UVB radiation.

Sample

Approximately five to six independent observations per assay as reported.

Duration

Up to 24 hours depending on assay.

Intervention

Hydrogen-rich culture medium after UVB exposure, with a PI3K inhibitor in mechanistic experiments.

Hydrogen form

H₂ dissolved in cell-culture medium.

H₂ specification

0.6 mmol/L dissolved H₂.

H₂ flow

Not applicable.

O₂ delivered with H₂

No oxygen was co-delivered.

Comparator

UVB-exposed and non-exposed conventional medium, with pathway-inhibitor comparisons.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Cell viability, ROS, oxidative markers, apoptosis and PI3K/Akt/FoxO signaling.

Reported result

Several viability, oxidative, apoptotic and pathway measures favored hydrogen-rich medium after UVB exposure.

Results-extraction completeness

The full article methods, intervention, outcomes, positive and null findings, funding and conflict statements were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Cell-only evidence; pathway-inhibitor experiments are mechanistic and do not establish patient benefit.

Applies directly to

The HaCaT cell model and tested exposure only.

Preliminary appraisal framework

In-vitro study appraisal — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Assay comparators were present, but replicate-level randomization, blinding and multiplicity control were not established.

Appraisal domains

Independent replicate reporting checked · Exposure and comparator checked · Assay blinding generally unreported · Multiplicity and selective reporting remain possible · External validity is limited to the tested cells

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 29532858 · DOI: 10.3892/ijmm.2018.3550

Publisher access

A free publisher or repository full article was checked.

Extraction basis

Publisher or repository full article and bibliographic record; checked 10 August 2026.

Record revision

2026-08-10