Hydrogenology
Hydrogenology editorial study record

Safety of inhaled hydrogen gas in healthy mice

Cole AR, Raza A, Ahmed H, Polizzotti BD, Padera RF, Andrews N, Kheir JN. · Medical Gas Research. 2019;9(3):133–138.

PreclinicalInhaled H₂Published 2019
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled blinded safety-screening experiment

Research topic

Other molecular hydrogen research

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Healthy ten-week-old female CD-1 mice exposed continuously in a gas-tight chamber.

Sample

50 mice: H₂ group n=25 and medical-air control n=25; blood tests and electron microscopy used smaller subsets of n=5 per group.

Duration

Continuous 72-hour exposure.

Intervention

Continuous chamber exposure to hydrogen diluted in medical air for 72 hours, with food and water available.

Hydrogen form

H₂ alone diluted in medical air — not Brown's gas.

H₂ specification

Source gas contained 3.2% H₂, 21% O₂ and balance N₂ and was titrated toward 2.4% chamber H₂. Mean measured exhaust concentration was 2.27% (95% CI 2.26–2.29%) and mean H₂-group total gas flow was 6.5±0.3 L/min.

H₂ flow

Approximately 148 mL/min H₂, calculated from the reported mean 6,500 mL/min total flow × mean measured 2.27% chamber H₂ concentration.

O₂ delivered with H₂

Approximately 21% O₂ in the medical-air carrier; approximately 1,365 mL/min O₂ calculated from 6,500 mL/min × 21%.

Comparator

Medical air at 3.0 L/min for the same 72-hour chamber exposure.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Survival, body weight, blinded SHIRPA neurobehavioral testing, spontaneous locomotor activity, complete blood count, serum chemistry, arterial blood gases, light microscopy of major organs and electron microscopy of airway epithelium.

Reported result

All mice survived. Body weight, total SHIRPA score, blood tests, blood gases and major-organ histology did not differ materially. Spontaneous locomotor activity fell in the H₂ group (p<0.0001), and two exposed mice had more prominent respiratory-epithelial secretory vesicles without structural injury; the authors considered the locomotor finding potentially artifactual but did not dismiss it.

Results-extraction completeness

The complete free article, source and measured gas concentrations, total flows, full safety panel, positive and null findings, limitations, funding and conflict statement were checked.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Female mice only, one concentration and duration, bloodwork in only five animals per group, many endpoints and unequal total gas flow between H₂ and control chambers. The authors calculated an 86% chance of at least one statistically significant result across the many endpoints. American Heart Association, NIH-supported core facilities and philanthropic donations funded the work; authors declared no related conflicts.

Applies directly to

A preclinical safety screen in healthy mice; it cannot establish long-term or patient safety, higher-dose safety or therapeutic benefit.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 31552876 · DOI: 10.4103/2045-9912.266988

Publisher access

Open-access PubMed Central article.

Extraction basis

Complete PubMed Central article and PubMed metadata; full-text extraction checked 9 August 2026.

Record revision

2026-08-10