Hydrogenology
Hydrogenology editorial study record

Hydrogen inhalation attenuates oxidative-stress-related endothelial injury after subarachnoid hemorrhage in rats

Zhuang Z et al. · Front Neurosci. 2020;13:1441.

PreclinicalInhaled H₂Published 2020
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Multi-part controlled rat subarachnoid-hemorrhage experiment comparing exposure durations and repeated dosing

Research topic

Other neurological conditions

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

182 male Sprague-Dawley rats with endovascular-perforation subarachnoid hemorrhage or sham surgery

Sample

182 rats across several experiments; principal comparisons commonly used n=6 per group

Duration

Single exposure or daily exposure for 3 or 7 days, depending on experiment

Intervention

3.3% H₂ inhalation beginning 30 minutes after hemorrhage for 30, 60 or 120 minutes; selected protocols were repeated daily

Hydrogen form

H₂ diluted in chamber air with oxygen adjusted separately — not Brown’s gas.

H₂ specification

3.3% H₂; chamber O₂ maintained at 21%.

H₂ flow

Not reported — chamber concentration is given, but H₂ flow in mL/min is not.

O₂ delivered with H₂

21% O₂ was maintained using supplemental oxygen; O₂ flow in mL/min was not reported.

Comparator

Room-air chamber exposure after hemorrhage and sham controls

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Brain edema, blood-brain barrier, oxidative and inflammatory markers, microthrombosis, vasospasm and short- and long-term neurological behavior

Reported result

The article reports lower vascular inflammatory and apoptotic markers, edema, microthrombi and vasospasm and improved neurological outcomes with selected H₂ schedules.

Results-extraction completeness

Gas composition, allocation structure, methods and principal results checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Large total animal number spread across many experiments, exposure schedules and molecular outcomes; no human participants.

Applies directly to

Experimental subarachnoid hemorrhage in rats.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 32038143 · DOI: 10.3389/fnins.2019.01441

Publisher access

Open-access full text is available through PMC and Frontiers.

Extraction basis

Open-access PMC/Frontiers full text and PubMed record.

Record revision

2026-08-10