Hydrogenology
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Hydrogen gas inhalation improves delayed brain injury by alleviating early brain injury after experimental subarachnoid hemorrhage

Kumagai K et al. · Sci Rep. 2020;10:12319.

PreclinicalInhaled H₂Published 2020
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled rat subarachnoid-hemorrhage plus carotid-occlusion experiment

Research topic

Other neurological conditions

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Rats with experimental subarachnoid hemorrhage and unilateral common-carotid occlusion

Sample

101 rats entered the protocol: sham n=31, control n=35 and H₂ n=33; later outcome cohorts were smaller

Duration

Two inhalation sessions with outcomes through day 7

Intervention

Inhalation on days 0 and 1: 2 hours on day 0 and 30 minutes on day 1

Hydrogen form

H₂ diluted in oxygen and nitrogen — not Brown’s gas.

H₂ specification

1.3% H₂, 30% O₂ and nitrogen balance.

H₂ flow

Not reported — concentration is given, but H₂ flow in mL/min is not.

O₂ delivered with H₂

30% O₂ was co-delivered; O₂ flow in mL/min was not reported.

Comparator

30% O₂ with nitrogen balance, without H₂, plus sham surgery

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Brain edema, neurological deficits, S100B, JNK, astrogliosis, vasospasm and neuronal death

Reported result

The article reports improvement in early and delayed brain-injury measures. Cerebral vasospasm did not improve significantly.

Results-extraction completeness

Gas composition, allocation, figures and positive and null results checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Complex animal model, attrition into smaller outcome cohorts, multiple laboratory endpoints and no human participants. The null vasospasm result is retained.

Applies directly to

Experimental subarachnoid hemorrhage with added carotid occlusion in rats, not patients with aneurysmal hemorrhage.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 32704088 · DOI: 10.1038/s41598-020-69028-5

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC full text and PubMed record.

Record revision

2026-08-10