Hydrogenology
Hydrogenology editorial study record

Controlled release of hydrogen by implantation of magnesium induces P53-mediated tumor cells apoptosis

Zan R, Wang H, Cai W, Ni J, Luthringer-Feyerabend BJC, Wang W, Peng H, Ji W, Yan J, Xia J, Song Y, Zhang X. · 2022.

PreclinicalOther formsPublished 2022
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled tumor-cell and mouse implantation study

Research topic

Cancer research

Administration classification

Other forms

Study result signal

Positive preclinical signal.

Outcome type

Tumor-growth, cell-death and mechanistic outcomes.

Reported in the source

Methods at a glance

Population or model

Human tumor cell lines and tumor-bearing mice exposed to locally released molecular hydrogen.

Sample

Cell experiments and small mouse groups; assay-specific counts are reported in the full article.

Duration

Article-specific cell time points and a 24-day mouse experiment.

Intervention

Magnesium implants released molecular hydrogen locally; separate cell experiments used controlled hydrogen exposure.

Hydrogen form

Locally generated molecular H₂ from implanted magnesium, with related in-vitro exposure.

H₂ specification

Article-specific implant and exposure conditions; systemic dose was not established.

H₂ flow

Not applicable.

O₂ delivered with H₂

No oxygen was intentionally co-delivered.

Comparator

Sham or non-hydrogen controls in cell and animal experiments.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Tumor growth, reactive-oxygen-species measures, apoptosis and p53-related signaling.

Reported result

The article reports reduced tumor growth and oxidative-stress measures and increased apoptosis-related signaling in the hydrogen-releasing conditions.

Results-extraction completeness

The full article was checked for methods, intervention details, outcomes, positive and null findings, funding and conflicts. This record does not imply medical efficacy.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical device-mediated delivery; magnesium and local implant effects cannot be assumed to equal inhaled or oral H₂ exposure.

Applies directly to

The tested cell and mouse tumor models only.

Preliminary appraisal framework

SYRCLE animal-study tool — preliminary

Preliminary risk-of-bias status

Some concerns

Appraisal rationale

Controlled experiments were reported, but allocation concealment, assessor blinding and multiplicity handling were unclear.

Appraisal domains

Sequence generation checked where reported · Allocation concealment generally unclear · Personnel blinding generally unclear · Outcome-assessor blinding generally unclear · No prospectively registered analysis plan identified

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 34820578 · DOI: 10.1016/j.bioactmat.2021.07.026

Publisher access

A free full article is available through PubMed Central.

Extraction basis

Official PubMed Central full article and PubMed bibliographic record; source identity, methods, intervention, results, funding and conflicts checked 10 August 2026.

Record revision

2026-08-10