Hydrogenology
Hydrogenology editorial study record

Hydrogen-rich water alleviates neuropathic pain through the modulation of bile acid/Takeda G-protein-coupled receptor 5-mediated autophagy

Zeng X et al. · Pain. 2026;167(3):663–677.

PreclinicalH₂-rich waterPublished 2026
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled mouse spinal-nerve-ligation experiments with dose response and mechanistic knockout studies

Research topic

Musculoskeletal and pain research

Administration classification

H₂-rich water

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Male and female mice with L4 spinal nerve ligation; additional TGR5 knockout and mechanistic groups

Sample

Principal behavioral and tissue comparisons report n=6 per group; the total number across all experiments was not stated as one figure

Duration

The full study used scheduled behavioral and tissue assessments after nerve ligation; one single duration does not describe all experiments

Intervention

Hydrogen-rich water administered by gavage after spinal nerve ligation

Hydrogen form

H₂ dissolved in water — H₂ only, not Brown’s gas.

H₂ specification

Dose-response experiments identified 3 ppm dissolved H₂ as the concentration used for subsequent experiments.

H₂ flow

Not applicable — oral gavage, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the intervention.

Comparator

Sham-operated and spinal-nerve-ligation mice receiving double-distilled water; additional mechanistic comparator groups

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Mechanical, heat and cold pain behaviors; spinal and dorsal-root microglia; gut barrier and microbiota; bile acids; TGR5 and autophagy markers

Reported result

The article reports dose-related improvement in pain behaviors, partial restoration of gut-barrier and microbiota measures, and loss or reduction of several effects in TGR5-manipulated experiments.

Results-extraction completeness

Dose, principal methods, results, supplemental links, conflicts and limitations checked in the open-access full text; the many experimental groups have not yet been extracted outcome by outcome.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Complex preclinical study with many experimental comparisons, microbiome and pathway analyses, group sizes commonly n=6 and no human participants. Fecal-transplant experiments and TGR5 overexpression were not performed; sex differences were not resolved.

Applies directly to

Male and female mouse models of nerve-ligation neuropathic pain, not people with chronic pain.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 41143867 · DOI: 10.1097/j.pain.0000000000003830

Publisher access

Open-access full text is available through PMC.

Extraction basis

Open-access PMC full text and PubMed bibliographic record.

Record revision

2026-08-10