Hydrogenology
Hydrogenology editorial study record

Intrathecal infusion of hydrogen-rich normal saline attenuates neuropathic pain via inhibition of activation of spinal astrocytes and microglia in rats

Ge Y et al. · PLoS One. 2014;9(5):e97436.

PreclinicalOther formsPublished 2014
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Rat L5 spinal-nerve-ligation experiments including crossover, repeated-treatment and preemptive-treatment cohorts

Research topic

Musculoskeletal and pain research

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Rats with L5 spinal-nerve ligation

Sample

Principal behavioral comparisons used n=10; immunostaining n=10 and selected protein assays n=5

Duration

Single-dose effect followed for 8 hours; repeated and preemptive schedules extended across the perioperative period

Intervention

20 μL hydrogen-rich saline injected intrathecally as single, repeated or preemptive treatment

Hydrogen form

H₂ dissolved in saline — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

0.6 mmol/L dissolved H₂; prepared under 0.4 MPa for 6 hours.

H₂ flow

Not applicable — intrathecal saline, not gas inhalation.

O₂ delivered with H₂

No O₂ was co-delivered as part of the H₂ intervention.

Comparator

Equal-volume normal saline, sham surgery and untreated catheterized controls

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Mechanical allodynia, thermal hyperalgesia, 8-OH-G, MnSOD nitration, glial activation, TNF-α and IL-1β

Reported result

The article reports short-acting analgesic behavior after one dose, improvement without observed tolerance during repeated dosing and lower oxidative, glial and inflammatory measures.

Results-extraction completeness

Concentration, volume, schedules, crossover design, positive findings, funding and conflicts checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Invasive intrathecal delivery in a small preclinical nerve-ligation model with several separate cohorts.

Applies directly to

Neuropathic pain behavior after nerve ligation in rats, not treatment of neuropathic pain in people.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 24857932 · DOI: 10.1371/journal.pone.0097436

Publisher access

Open-access full text is available through PMC and PLOS.

Extraction basis

Open-access PMC/PLOS full text and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10