Hydrogenology
Hydrogenology editorial study record

Hydrogen gas represses the progression of lung cancer via down-regulating CD47

Meng J, Liu L, Wang D, Yan Z, Chen G. · Bioscience Reports. 2020;40(4):BSR20192761.

PreclinicalInhaled H₂Published 2020
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Mechanistic lung-cancer cell study with archival xenograft tissues from a prior mouse experiment

Research topic

Cancer research

Administration classification

Inhaled H₂

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Human A549 and H1975 non-small-cell-lung-cancer cell lines, macrophage co-cultures and previously collected A549 xenograft tissues from male nude mice.

Sample

Cell experiments were performed in triplicate. The mouse-tissue group sizes were not reported in this article because the tissues came from a previous experiment; no total is inferred.

Duration

Cells were assessed from 12 to 72 hours depending on assay; prior mice received two hours/day for four weeks.

Intervention

Cells received 20%, 40% or 60% H₂ for outcome-specific periods. The prior mouse experiment used 60% H₂ inhalation for two hours daily over four weeks.

Hydrogen form

High-concentration H₂ gas; the carrier composition was not reported, so the mixture is not classified as Brown's gas.

H₂ specification

20%, 40% and 60% H₂ in vitro; 60% H₂ for mice. Total flow and carrier-gas composition were absent.

H₂ flow

Not reported — total gas flow is absent.

O₂ delivered with H₂

Carrier O₂ concentration and flow were not reported and are not inferred.

Comparator

Standard culture conditions, CD47 knockdown/overexpression controls, atmosphere-exposed xenograft control tissues and cisplatin comparator tissues from the prior experiment.

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Cell proliferation, migration, invasion, apoptosis, CD47/CDC42 signaling, macrophage phagocytosis and xenograft histology/immunohistochemistry.

Reported result

H₂ reduced several malignant cell behaviors and CD47/CDC42 measures and increased apoptosis/phagocytosis. CD47 overexpression weakened, and CD47 knockdown strengthened, selected H₂-associated effects. Mouse-tissue findings were supportive but did not constitute a newly randomized in-vivo experiment in this article.

Results-extraction completeness

The complete free PMC article was checked for cell concentrations/timing, genetic comparisons, archival-animal provenance, missing mouse sample and gas-flow/carrier data, results, funding and conflicts.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Mostly cell-line mechanism work; mouse samples and complete prior allocation are absent here, the 60% gas carrier/flow is unspecified, multiple endpoints were run in triplicate and clinical antitumour efficacy was not tested. The work was funded by a hydrogen-in-lung-cancer translational program; authors declared no competing interests and named a commercial gas-machine supplier.

Applies directly to

Lung-cancer cell lines and archival mouse xenograft tissue; it does not establish treatment of human lung cancer.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 32314789 · DOI: 10.1042/BSR20192761

Publisher access

Free full article in PubMed Central.

Extraction basis

Complete PMC article, cited provenance of the mouse tissues and PubMed metadata; full-text extraction checked 9 August 2026.

Record revision

2026-08-10