Hydrogenology
Hydrogenology editorial study record

Drinking Hydrogen-Rich Water Alleviates Chemotherapy-Induced Neuropathic Pain Through the Regulation of Gut Microbiota

Lian N et al. · J Pain Res. 2021;14:681–691.

PreclinicalH₂-rich waterPublished 2021
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Randomized four-group mouse oxaliplatin-neuropathy experiment with behavioral, microbiome and molecular analyses

Research topic

Musculoskeletal and pain research

Administration classification

H₂-rich water

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Female C57BL/6J mice receiving oxaliplatin or saline

Sample

Behavior n=10/group, microbiome n=6/group and biochemical assays n=5/group

Duration

20 days; oxaliplatin 3 mg/kg daily on days 1–5

Intervention

Hydrogen-rich drinking water ad libitum throughout the 20-day experiment

Hydrogen form

H₂ dissolved in drinking water — H₂ only, not Brown’s gas and not inhalation.

H₂ specification

Water was maintained at about 800–1,000 ppb dissolved H₂ and replaced daily.

H₂ flow

Water preparation used pure H₂ at 400 mL/min for 10 minutes, then 100 mL/min continuously; this was preparation flow, not inhaled animal dose.

O₂ delivered with H₂

No O₂ was co-delivered as part of the drinking-water intervention.

Comparator

Ordinary drinking water in saline- and oxaliplatin-treated mice

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Mechanical paw-withdrawal threshold, gut-microbiota diversity and composition, cytokines, oxidative markers, LPS and TLR4

Reported result

The article reports less mechanical hyperalgesia and changes in microbiota and LPS–TLR4-related measures. Several inflammatory and oxidative differences were observed in dorsal-root ganglia but not spinal cord or serum.

Results-extraction completeness

Allocation, H₂ preparation flow and concentration, main positive and null tissue findings, funding and limitations checked in the open-access full text.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Preclinical chemotherapy-toxicity model using only female mice, multiple microbiome and molecular outcomes, and no fecal-transplant test of causality.

Applies directly to

Oxaliplatin-associated pain behavior in mice, not neuropathy prevention in cancer patients.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 33732014 · DOI: 10.2147/JPR.S288289

Publisher access

Open-access full text is available through PMC and the publisher.

Extraction basis

Open-access PMC/publisher full text and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10