Hydrogenology
Hydrogenology editorial study record

Hydrogen attenuates postoperative pain through Trx1/ASK1/MMP9 signaling pathway

Li J et al. · J Neuroinflammation. 2023;20(1):22.

PreclinicalOther formsPublished 2023
Study record, not medical adviceThis page reports one source and Hydrogenology’s source-text extraction. It does not establish that molecular hydrogen is effective, safe or appropriate for any person. Human, animal and laboratory evidence must not be treated as equivalent.
Source and classification

What kind of evidence is this?

Evidence stream

Preclinical

Reported design

Controlled mouse plantar-incision experiments with genetic and pharmacologic pathway comparisons plus BV-2 cell experiments

Research topic

Musculoskeletal and pain research

Administration classification

Other forms

Study result signal

Not reported in this record.

Outcome type

Not reported in this record.

Reported in the source

Methods at a glance

Population or model

Mice with plantar-incision postoperative pain and cultured BV-2 microglial cells

Sample

Behavioral cohorts commonly used n=12 and mechanistic assays n=4–6; several separate cohorts were used

Duration

Twice-daily treatment with spinal and behavioral assessment at 24 hours and day 5

Intervention

Hydrogen-rich saline 5 mL/kg intraperitoneally twice daily in mice; H₂-rich medium in the cell experiments

Hydrogen form

H₂ dissolved in saline or culture medium — not Brown’s gas and not gas inhalation in the in-vivo treatment.

H₂ specification

5 mL/kg H₂-rich saline twice daily; numeric dissolved-H₂ concentration was not located in the extracted methods.

H₂ flow

Not applicable to the mice — intraperitoneal saline, not inhalation. Cell-medium preparation flow was not reported.

O₂ delivered with H₂

No O₂ was co-delivered as part of the H₂ intervention.

Comparator

Sham and plantar-incision groups receiving vehicle, plus MMP-9 knockout and ASK1/Trx1 inhibitor comparisons

Reported, not endorsed

Outcomes and reported result

Outcomes measured

Mechanical allodynia, spinal Trx1/ASK1/p38/JNK signaling, MMP-9, cytokines and microglial activation

Reported result

The article reports less mechanical allodynia, MMP-9 activity, inflammatory signaling and microglial activation with H₂-rich saline; a Trx1 inhibitor abolished several reported effects.

Results-extraction completeness

Dose, principal cohorts, mechanistic comparisons, figures, funding and conflicts checked in the open-access full text; numeric dissolved-H₂ concentration remains unconfirmed.

A reported association, difference or mechanism is not automatically a clinical benefit. Null findings, outcome type, study design and precision all matter.

Interpretation limits

Cautions and applicability

Design and reporting cautions

Multiple preclinical cohorts and mechanistic interventions; no human participants and no clinical postoperative-pain endpoint.

Applies directly to

Experimental plantar-incision pain in mice, not postoperative pain treatment in people.

Preliminary appraisal framework

Not reported in this record.

Preliminary risk-of-bias status

Not reported in this record.

Appraisal rationale

Not reported in this record.

No single-study GRADE certainty rating is assigned. Certainty is assessed by important outcome across a complete eligible evidence set, not by attaching a final grade to one source.

Provenance

Sources and record status

Identifiers

PMID: 36737785 · DOI: 10.1186/s12974-022-02670-0

Publisher access

Open-access full text is available through PMC and the publisher.

Extraction basis

Open-access PMC/publisher full text and PubMed record; no retraction or expression of concern was shown in the checked records on 7 August 2026.

Record revision

2026-08-10